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標(biāo)記(jì)一抗(kàng)

FITC標記的載(zǎi)脂蛋白(bái)A2抗體(tǐ)

文(wén)字:[大(dà)][中][小(xiǎo)] 2017-5-3    瀏(liú)覽(lǎn)次數(shù):2010    

                                   FITC標(biāo)記(jì)的(dí)載脂(zhī)蛋白(bái)A2抗(kàng)體                                                                                                                                                
英文(wén)名(míng)稱Anti-APOA2/FITC
中(zhōng)文名稱(chēng):FITC標(biāo)記的載(zǎi)脂(zhī)蛋(dàn)白(bái)A2抗體
別(bié)    名APO A2; Apo AII; Apo-AII; APOA 2; ApoA II; ApoA-II; APOA2; APOA2_HUMAN; APOAII; Apolipoprotein A II; Apolipoprotein A-II(1-76); Apolipoprotein A2; Apolipoprotein AII; ApolipoproteinA II; OTTHUMP.  

詳細(xì)介紹:


規(guī)格(gé):100ul 
說(shuō) 明 書100ul  
研(yán)究領(lǐng)域腫瘤  心血(xiě)管  細胞(bāo)生(shēng)物  免疫(yì)學(xué)  信號轉導(dǎo)  新陳代謝  
抗體來源(yuán)Rabbit
克(kè)隆(lóng)類(lèi)型Polyclonal
交叉反應 Human, Mouse, Rat, Dog, 
產品應用IF=1:50-200  
not yet tested in other applications.
optimal dilutions/concentrations should be determined by the end user.
分 子 量(liáng)9kDa
性    狀(zhuàng)Lyophilized or Liquid
濃    度(dù)1mg/ml
免(miǎn) 疫(yì) 原KLH conjugated synthetic peptide derived from human APOA2
亞    型(xíng)IgG
純化方法affinity purified by Protein A
儲 存(cún) 液(yè)0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.
保(bǎo)存(cún)條件(jiàn)Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

相關資料(liào):


產品(pǐn)介紹background:
Apolipoprotein A II is the second most abundant protein of the high density lipoprotein particles. The apolipoprotein A II gene consists of 4 exons and 3 introns. The four exons encode the 5' untranslated region, pre peptide, a short N terminal domain and a C terminal domain composed of a variable number of lipid binding amphipathic helices. Familial apolipoprotein A II deficiency may result from a splice junction alteration which blocks splicing of intron 3 from the primary transcript and result in the formation of a non functional mRNA.

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